CYCLO TREN™ MAY HELP TO:
- Optimize Testosterone utilization*
- ENHANCE LH LEVELS*
- HELPS minimize estrogen impact on the body*
- promote LEAN Muscle AND Performance GAINS*
Infinite Labs® Cyclo Tren™ is the optimal testosterone-enhancing, hypertrophic supplementation to integrate into any cutting training regimen. Cyclo Tren™ introduces three scientifically-designed matrixes to provide you with that extra boost needed to promote anabolic effects by supporting luteinizing hormone release and testosterone utilization, while simultaneously incorporating a lipid-incinerating component to help any athlete reach their goal of cutting down.*
Anabolic Activation Catalyst
Cyclo Tren™ combines testosterone-supporting amino acid, D-aspartic acid (DAA) with the potent, natural extract Eurycoma Longifolia Jack to stimulate the production of endogenous testosterone and to reduce the levels of bound and metabolically inactive testosterone in the body.* It enhances the cellular production of signal molecules that control the secretion of Luteinizing Hormone (LH) and testosterone to provide you with the anabolic environment necessary for muscles to optimally develop and grow. In combination with Fenugreek that supports endogenous testosterone levels, Cyclo Tren™ offers the ideal arrangement of components to ultimately maximize your strength gains.* Bioperine® is also incorporated into this stack to enhance the bioavailability of these ingredients via increased absorption rates.*
Lean Mass Amplification
L-Carnitine plays an essential role in energy production and increases the rate at which fat is burned; L-Carnitine Tartrate makes a varied contribution, directly and indirectly on normal functioning of the metabolism. L-Carnitine Tartrate supplementation has been evaluated in resistance exercise-trained humans as an enhancer of the hormonal responses to resistance exercise and as a recovery promoter.* 3-weeks supplementation with L-Carnitine Tartrate is suggested to reduce muscle damage produced by an acute bout of high-intensity resistance exercise in two cross-over, placebo-controlled trials.* Researchers suggest that less muscle damage may have resulted due to more available hormonal receptors for binding interactions with anabolic hormones. With more hormonal receptors available for binding interactions with anabolic hormones, muscle development is potentially increased.* This matrix also includes Coleus Forskohlii which activates cAMP, thus promoting a number of chemical reactions including the breakdown of fats. Moreover, cAMP has been suggested to stimulate another enzyme known as hormone sensitive lipase (LIPE). LIPE is activated as the body mobilizes energy stores; it does so by hydrolyzing triglycerides to free fatty acids in the adipose tissue. In combination with caffeine, this matrix gives Cyclo Tren™ its fat-targeting capability to ensure your body metabolizes lipids as efficiently as possible.*
In order to promote optimal testosterone utilization, Cyclo Tren™ combines an estrogen-blocking matrix into its unique formula to minimize any negative effects estrogen may have on the anabolic process. Vitex Agnus Castus is a fruit-plant that increases the natural production of testosterone, while lowering prolactin levels. In combination with Diindolylmethane (DIM), this matrix promotes beneficial estrogen metabolism in both sexes by reducing the levels of 16-hydroxy estrogen metabolites and increasing the formation of 2-hydroxy estrogen metabolites. In other words, DIM converts active estrogens into inactive estrogen metabolites, thus freeing up the testosterone molecule by separating them from the testosterone-binding proteins; this ultimately promotes the circulation of free testosterone.*
Ang HH & Cheang HS (2001). Effects of Eurycoma longifolia jack on laevator ani muscle in both uncastrated and testosterone-stimulated castrated intact male rats. Pharmacal Research. 24 (5): 437–40.
D’Aniello A, Di Fiore MM, D’Aniello G, Colin FE, Lewis G & Setchell BP (1998). Secretion of D-aspartic acid by the rat testis and its role in endocrinology of the testis and spermatogenesis. FEBS Lett. 25:436(1):23-7.
D’Aniello A (2007). D-Aspartic acid: An endogenous amino acid with an important neuroendocrine role. Brain Res Rev. 53(2):215-34.
Kraemer, WJ, et al. (2006). Androgenic Responses to resistance exercise: Effects of feeding and L-Carnitine. Med Sci Sports Exerc. 38(7): 1288-1296.
Kraemer, WJ, et al. (2003). The Effects of L-Carnitine L-Tartrate supplementation on hormonal responses to resistance exercise and recovery. J Strength Cond Res. 17(3): 455-462.
Miyake K, Tezuka Y, Awale S, Li F & Kadota S (2009). Quassinoids from Eurycoma longifolia. Journal of Natural Products. 72 (12): 2135–40.
Ratamess, NA, et al. (2005). Androgen receptor content following heavy resistance exercise in men. J Steroid Biochem Mol Biol 93. (1): 35-42.
Srivasta SK, Khatoon CS & Mehrotra SR (2002). Pharmacognistic evaluation of coleus forskohlii. Pharmaceutical Biology. 40:129–134.
Topo E, Soricelli A, D’Aniello A, Ronsini S & D’Aniello G (2009). The role and molecular mechanism of D-aspartic acid in the release and synthesis of LH and testosterone in humans and rats. Reprod Biol Endocrinol. 7:120.
Volek, JS, et al. (2002). L-Carnitine L-Tartrate supplementation favorably affects markers of recovery from exercise stress. Am J Physiol Endocrinol Metab. 282(2): E474-E482.
Willoughby, DS & L. Taylor (2004). Effects of sequential bouts of resistance exercise on androgen receptor expression. Med Sci Sports Exerc. 36(9):1499-1506.